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Editorial: Lessons From the Classic Scientific Literature.

Molecular endocrinology (Baltimore, Md.)

Mirmira RG.
PMID: 26426488
Mol Endocrinol. 2015 Oct;29(10):1385-7. doi: 10.1210/me.2015-1231.

No abstract available.

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Mirmira RG. Editorial: Lessons From the Classic Scientific Literature. Mol Endocrinol. 2015;29(10):1385-7doi: 10.1210/me.2015-1231.
Mirmira, R. G. (2015). Editorial: Lessons From the Classic Scientific Literature. Molecular endocrinology (Baltimore, Md.), 29(10), 1385-7. https://doi.org/10.1210/me.2015-1231
Mirmira, Raghavendra G. "Editorial: Lessons From the Classic Scientific Literature." Molecular endocrinology (Baltimore, Md.) vol. 29,10 (2015): 1385-7. doi: https://doi.org/10.1210/me.2015-1231
Mirmira RG. Editorial: Lessons From the Classic Scientific Literature. Mol Endocrinol. 2015 Oct;29(10):1385-7. doi: 10.1210/me.2015-1231. PMID: 26426488; PMCID: PMC4588731.
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The importance of basic science and research training for the next generation of physicians and physician scientists.

Molecular endocrinology (Baltimore, Md.)

DeFranco DB, Sowa G.
PMID: 25437019
Mol Endocrinol. 2014 Dec;28(12):1919-21. doi: 10.1210/me.2014-1343.

No abstract available.

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DeFranco DB, Sowa G. The importance of basic science and research training for the next generation of physicians and physician scientists. Mol Endocrinol. 2014;28(12):1919-21doi: 10.1210/me.2014-1343.
DeFranco, D. B., & Sowa, G. (2014). The importance of basic science and research training for the next generation of physicians and physician scientists. Molecular endocrinology (Baltimore, Md.), 28(12), 1919-21. https://doi.org/10.1210/me.2014-1343
DeFranco, Donald B, and Sowa, Gwendolyn. "The importance of basic science and research training for the next generation of physicians and physician scientists." Molecular endocrinology (Baltimore, Md.) vol. 28,12 (2014): 1919-21. doi: https://doi.org/10.1210/me.2014-1343
DeFranco DB, Sowa G. The importance of basic science and research training for the next generation of physicians and physician scientists. Mol Endocrinol. 2014 Dec;28(12):1919-21. doi: 10.1210/me.2014-1343. PMID: 25437019; PMCID: PMC5414780.
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Corrigendum. Impaired fertility and FSH synthesis in gonadotrope-specific Foxl2 knockout mice.

Molecular endocrinology (Baltimore, Md.)

[No authors listed]
PMID: 25723485
Mol Endocrinol. 2015 Mar;29(3):484. doi: 10.1210/me.2015-1023.

No abstract available.

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Corrigendum. Impaired fertility and FSH synthesis in gonadotrope-specific Foxl2 knockout mice. Mol Endocrinol. 2015;29(3):484doi: 10.1210/me.2015-1023.
(2015). Corrigendum. Impaired fertility and FSH synthesis in gonadotrope-specific Foxl2 knockout mice. Molecular endocrinology (Baltimore, Md.), 29(3), 484. https://doi.org/10.1210/me.2015-1023
"Corrigendum. Impaired fertility and FSH synthesis in gonadotrope-specific Foxl2 knockout mice." Molecular endocrinology (Baltimore, Md.) vol. 29,3 (2015): 484. doi: https://doi.org/10.1210/me.2015-1023
Corrigendum. Impaired fertility and FSH synthesis in gonadotrope-specific Foxl2 knockout mice. Mol Endocrinol. 2015 Mar;29(3):484. doi: 10.1210/me.2015-1023. PMID: 25723485; PMCID: PMC4347285.
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Retraction. Orphan receptor small heterodimer partner is an important mediator of glucose homeostasis.

Molecular endocrinology (Baltimore, Md.)

Wang L, Huang J, Saha P, Kulkarni RN, Hu M, Kim Y, Park K, Chan L, Rajan AS, Lee I, Moore DD.
PMID: 18757780
Mol Endocrinol. 2008 Sep;22(9):2203. doi: 10.1210/mend.22.9.9999.

No abstract available.

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Wang L, Huang J, Saha P, et al. Retraction. Orphan receptor small heterodimer partner is an important mediator of glucose homeostasis. Mol Endocrinol. 2008;22(9):2203doi: 10.1210/mend.22.9.9999.
Wang, L., Huang, J., Saha, P., Kulkarni, R. N., Hu, M., Kim, Y., Park, K., Chan, L., Rajan, A. S., Lee, I., & Moore, D. D. (2008). Retraction. Orphan receptor small heterodimer partner is an important mediator of glucose homeostasis. Molecular endocrinology (Baltimore, Md.), 22(9), 2203. https://doi.org/10.1210/mend.22.9.9999
Wang, L, et al. "Retraction. Orphan receptor small heterodimer partner is an important mediator of glucose homeostasis." Molecular endocrinology (Baltimore, Md.) vol. 22,9 (2008): 2203. doi: https://doi.org/10.1210/mend.22.9.9999
Wang L, Huang J, Saha P, Kulkarni RN, Hu M, Kim Y, Park K, Chan L, Rajan AS, Lee I, Moore DD. Retraction. Orphan receptor small heterodimer partner is an important mediator of glucose homeostasis. Mol Endocrinol. 2008 Sep;22(9):2203. doi: 10.1210/mend.22.9.9999. PMID: 18757780; PMCID: PMC5419458.
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Steroids and miRNAs in assessment of ovarian tissue damage following cryopreservation.

Journal of molecular endocrinology

Islam N, Ugwoke SP, Alhamdan R, Medrano JH, Campbell BK, Marsters P, Maalouf WE.
PMID: 30925460
J Mol Endocrinol. 2019 Mar 01; doi: 10.1530/JME-18-0237. Epub 2019 Mar 01.

Ovarian cortical tissue cryopreservation is a relatively novel approach to preserving fertility in women diagnosed with cancer. However, the effects of freezing-thawing are not fully understood, mainly due to the lack of suitable methods to assess tissue's survival after...

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Islam N, Ugwoke SP, Alhamdan R, et al. Steroids and miRNAs in assessment of ovarian tissue damage following cryopreservation. J Mol Endocrinol. 2019;doi: 10.1530/JME-18-0237.
Islam, N., Ugwoke, S. P., Alhamdan, R., Medrano, J. H., Campbell, B. K., Marsters, P., & Maalouf, W. E. (2019). Steroids and miRNAs in assessment of ovarian tissue damage following cryopreservation. Journal of molecular endocrinology, . https://doi.org/10.1530/JME-18-0237
Islam, Nadia, et al. "Steroids and miRNAs in assessment of ovarian tissue damage following cryopreservation." Journal of molecular endocrinology vol. (2019). doi: https://doi.org/10.1530/JME-18-0237
Islam N, Ugwoke SP, Alhamdan R, Medrano JH, Campbell BK, Marsters P, Maalouf WE. Steroids and miRNAs in assessment of ovarian tissue damage following cryopreservation. J Mol Endocrinol. 2019 Mar 01; doi: 10.1530/JME-18-0237. Epub 2019 Mar 01. PMID: 30925460.
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Thyroid Function Disruptors: from nature to chemicals.

Journal of molecular endocrinology

Oliveira KJ, Chiamolera MI, Giannocco G, Pazos-Moura CC, Ortiga-Carvalho TM.
PMID: 30006341
J Mol Endocrinol. 2018 Jul 13; doi: 10.1530/JME-18-0081. Epub 2018 Jul 13.

The modern concept of thyroid disruptors includes man-made chemicals and bioactive compounds from food that interfere with any aspect of the hypothalamus-pituitary-thyroid axis, thyroid hormone biosynthesis and secretion, blood and transmembrane transport, metabolism and local action of thyroid hormones....

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Oliveira KJ, Chiamolera MI, Giannocco G, et al. Thyroid Function Disruptors: from nature to chemicals. J Mol Endocrinol. 2018;doi: 10.1530/JME-18-0081.
Oliveira, K. J., Chiamolera, M. I., Giannocco, G., Pazos-Moura, C. C., & Ortiga-Carvalho, T. M. (2018). Thyroid Function Disruptors: from nature to chemicals. Journal of molecular endocrinology, . https://doi.org/10.1530/JME-18-0081
Oliveira, Karen J, et al. "Thyroid Function Disruptors: from nature to chemicals." Journal of molecular endocrinology vol. (2018). doi: https://doi.org/10.1530/JME-18-0081
Oliveira KJ, Chiamolera MI, Giannocco G, Pazos-Moura CC, Ortiga-Carvalho TM. Thyroid Function Disruptors: from nature to chemicals. J Mol Endocrinol. 2018 Jul 13; doi: 10.1530/JME-18-0081. Epub 2018 Jul 13. PMID: 30006341.
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Integrated Omics: Tools, Advances, and Future Approaches.

Journal of molecular endocrinology

Misra BB, Langefeld CD, Olivier M, Cox LA.
PMID: 30006342
J Mol Endocrinol. 2018 Jul 13; doi: 10.1530/JME-18-0055. Epub 2018 Jul 13.

With the rapid adoption of high-throughput omic approaches to analyze biological samples such as genomics, transcriptomics, proteomics, and metabolomics, each analysis can generate tera- to peta-byte sized data files on a daily basis. These data file sizes, together with...

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Misra BB, Langefeld CD, Olivier M, et al. Integrated Omics: Tools, Advances, and Future Approaches. J Mol Endocrinol. 2018;doi: 10.1530/JME-18-0055.
Misra, B. B., Langefeld, C. D., Olivier, M., & Cox, L. A. (2018). Integrated Omics: Tools, Advances, and Future Approaches. Journal of molecular endocrinology, . https://doi.org/10.1530/JME-18-0055
Misra, Biswapriya B, et al. "Integrated Omics: Tools, Advances, and Future Approaches." Journal of molecular endocrinology vol. (2018). doi: https://doi.org/10.1530/JME-18-0055
Misra BB, Langefeld CD, Olivier M, Cox LA. Integrated Omics: Tools, Advances, and Future Approaches. J Mol Endocrinol. 2018 Jul 13; doi: 10.1530/JME-18-0055. Epub 2018 Jul 13. PMID: 30006342.
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Role of AMPK in the protective effects exerted by triiodothyronine in ischemic-reperfused myocardium.

Journal of molecular endocrinology

Hermann R, Mestre Cordero VE, Fernández Pazos MLM, Córdoba MF, Reznik FJ, Vélez DE, Fellet AL, Marina Prendes MG.
PMID: 33640872
J Mol Endocrinol. 2021 Mar;66(3):207-221. doi: 10.1530/JME-20-0314.

Recent studies have provided evidence that triiodothyronine (T3) might play an effective role in the recovery of ischemic myocardium, through the preservation of mitochondrial function and the improvement of energy substrate metabolism. To this respect, it has been suggested...

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Hermann R, Mestre Cordero VE, Fernández Pazos MLM, et al. Role of AMPK in the protective effects exerted by triiodothyronine in ischemic-reperfused myocardium. J Mol Endocrinol. 2021;66(3):207-221doi: 10.1530/JME-20-0314.
Hermann, R., Mestre Cordero, V. E., Fernández Pazos, M. L. M., Córdoba, M. F., Reznik, F. J., Vélez, D. E., Fellet, A. L., & Marina Prendes, M. G. (2021). Role of AMPK in the protective effects exerted by triiodothyronine in ischemic-reperfused myocardium. Journal of molecular endocrinology, 66(3), 207-221. https://doi.org/10.1530/JME-20-0314
Hermann, Romina, et al. "Role of AMPK in the protective effects exerted by triiodothyronine in ischemic-reperfused myocardium." Journal of molecular endocrinology vol. 66,3 (2021): 207-221. doi: https://doi.org/10.1530/JME-20-0314
Hermann R, Mestre Cordero VE, Fernández Pazos MLM, Córdoba MF, Reznik FJ, Vélez DE, Fellet AL, Marina Prendes MG. Role of AMPK in the protective effects exerted by triiodothyronine in ischemic-reperfused myocardium. J Mol Endocrinol. 2021 Mar;66(3):207-221. doi: 10.1530/JME-20-0314. PMID: 33640872.
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The dual role of RFX6 in directing β cell development and insulin production.

Journal of molecular endocrinology

Lu J, Cheng C, Cheng ZC, Wu Q, Shen H, Yuan MX, Zhang B, Yang JK.
PMID: 33350979
J Mol Endocrinol. 2021 Feb;66(2):129-140. doi: 10.1530/JME-20-0119.

RFX6 transcription factor is believed to play a central role in directing cell development of insulin-producing pancreatic islet. RFX6 homozygous mutations cause syndromic neonatal diabetes with hypoplastic pancreas. However, RFX6 heterozygous mutations cause maturity-onset diabetes of the young (MODY)...

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Lu J, Cheng C, Cheng ZC, et al. The dual role of RFX6 in directing β cell development and insulin production. J Mol Endocrinol. 2021;66(2):129-140doi: 10.1530/JME-20-0119.
Lu, J., Cheng, C., Cheng, Z. C., Wu, Q., Shen, H., Yuan, M. X., Zhang, B., & Yang, J. K. (2021). The dual role of RFX6 in directing β cell development and insulin production. Journal of molecular endocrinology, 66(2), 129-140. https://doi.org/10.1530/JME-20-0119
Lu, Jing, et al. "The dual role of RFX6 in directing β cell development and insulin production." Journal of molecular endocrinology vol. 66,2 (2021): 129-140. doi: https://doi.org/10.1530/JME-20-0119
Lu J, Cheng C, Cheng ZC, Wu Q, Shen H, Yuan MX, Zhang B, Yang JK. The dual role of RFX6 in directing β cell development and insulin production. J Mol Endocrinol. 2021 Feb;66(2):129-140. doi: 10.1530/JME-20-0119. PMID: 33350979.
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GCgx: transcriptome-wide exploration of the response to glucocorticoids.

Journal of molecular endocrinology

Cao Q, Boo Irizarry Y, Yazhuk S, Tran T, Gadkari M, Franco LM.
PMID: 34787097
J Mol Endocrinol. 2021 Dec 10;68(2):B1-B4. doi: 10.1530/JME-21-0107.

Glucocorticoids are the cornerstone of immunosuppressive and anti-inflammatory therapy in humans, yet the mechanisms of glucocorticoid immunoregulation and toxicity remain unclear. The response to glucocorticoids is highly cell type-dependent, so translating results from different experimental systems into a better...

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Cao Q, Boo Irizarry Y, Yazhuk S, et al. GCgx: transcriptome-wide exploration of the response to glucocorticoids. J Mol Endocrinol. 2021;68(2):B1-B4doi: 10.1530/JME-21-0107.
Cao, Q., Boo Irizarry, Y., Yazhuk, S., Tran, T., Gadkari, M., & Franco, L. M. (2021). GCgx: transcriptome-wide exploration of the response to glucocorticoids. Journal of molecular endocrinology, 68(2), B1-B4. https://doi.org/10.1530/JME-21-0107
Cao, Qilin, et al. "GCgx: transcriptome-wide exploration of the response to glucocorticoids." Journal of molecular endocrinology vol. 68,2 (2021): B1-B4. doi: https://doi.org/10.1530/JME-21-0107
Cao Q, Boo Irizarry Y, Yazhuk S, Tran T, Gadkari M, Franco LM. GCgx: transcriptome-wide exploration of the response to glucocorticoids. J Mol Endocrinol. 2021 Dec 10;68(2):B1-B4. doi: 10.1530/JME-21-0107. PMID: 34787097; PMCID: PMC8691098.
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FGF23 signalling and physiology.

Journal of molecular endocrinology

Ho BB, Bergwitz C.
PMID: 33338030
J Mol Endocrinol. 2021 Feb;66(2):R23-R32. doi: 10.1530/JME-20-0178.

Fibroblast growth factor 23 (FGF23) is a phosphotropic hormone that belongs to a subfamily of endocrine FGFs with evolutionarily conserved functions in worms and fruit flies. FAM20C phosphorylates FGF23 post-translationally, targeting it to proteolysis through subtilisin-like proprotein convertase FURIN,...

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Ho BB, Bergwitz C. FGF23 signalling and physiology. J Mol Endocrinol. 2021;66(2):R23-R32doi: 10.1530/JME-20-0178.
Ho, B. B., & Bergwitz, C. (2021). FGF23 signalling and physiology. Journal of molecular endocrinology, 66(2), R23-R32. https://doi.org/10.1530/JME-20-0178
Ho, Bryan B, and Bergwitz, Clemens. "FGF23 signalling and physiology." Journal of molecular endocrinology vol. 66,2 (2021): R23-R32. doi: https://doi.org/10.1530/JME-20-0178
Ho BB, Bergwitz C. FGF23 signalling and physiology. J Mol Endocrinol. 2021 Feb;66(2):R23-R32. doi: 10.1530/JME-20-0178. PMID: 33338030.
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Heterogeneity of G protein activation by the calcium-sensing receptor.

Journal of molecular endocrinology

Abid HA, Inoue A, Gorvin CM.
PMID: 34077389
J Mol Endocrinol. 2021 Jun 21;67(2):41-53. doi: 10.1530/JME-21-0058.

The calcium-sensing receptor (CaSR) is a G protein-coupled receptor that plays a fundamental role in extracellular calcium (Ca2+e) homeostasis by regulating parathyroid hormone release and urinary calcium excretion. The CaSR has been described to activate all four G protein...

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Abid HA, Inoue A, Gorvin CM. Heterogeneity of G protein activation by the calcium-sensing receptor. J Mol Endocrinol. 2021;67(2):41-53doi: 10.1530/JME-21-0058.
Abid, H. A., Inoue, A., & Gorvin, C. M. (2021). Heterogeneity of G protein activation by the calcium-sensing receptor. Journal of molecular endocrinology, 67(2), 41-53. https://doi.org/10.1530/JME-21-0058
Abid, Hasnat Ali, et al. "Heterogeneity of G protein activation by the calcium-sensing receptor." Journal of molecular endocrinology vol. 67,2 (2021): 41-53. doi: https://doi.org/10.1530/JME-21-0058
Abid HA, Inoue A, Gorvin CM. Heterogeneity of G protein activation by the calcium-sensing receptor. J Mol Endocrinol. 2021 Jun 21;67(2):41-53. doi: 10.1530/JME-21-0058. PMID: 34077389; PMCID: PMC8240730.
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Showing 1 to 12 of 107 entries